Sebaceous Cyst – Epidermoid Disorder

What are Sebaceous Cyst?

The problem of sebaceous cyst is very common among the females especially the girls. They occur due to various skin issues including the damage to the follicles. They are also become a result of excessive use of cosmetics and beauty products made of oils. The Sebaceous Cyst Removal are not harmful in nature however they create severe results if left in treated for longer. They can become a reason for skin cancer because of the blockage and rupture.

Sebaceous Cyst Causes

They are composed of yellow or white colored mass that excretes out upon any kind of rupture. Sometimes they are hard and have a black head end. The cysts can be formed on any skin surface where as on the dark complexion the sebaceous cyst are pigmented.

Sebaceous Cyst Symptoms

Usually people mix the Sebaceous Cyst Symptoms with epidermoid cysts and pilar cysts. They are of two different kinds and have different nature from the sebaceous cyst. The epidermoid cysts are present in the upper layer of the skin called epidermis where as the pilar cysts are caused due to the hair follicles. The sebaceous cyst do not rise from the sebaceous glands they are composed of sebum and are rare. The other ones are composed of keratin.

Sebaceous Cyst

Sebaceous Cyst Diagnosis

Effects of different types of sebaceous cyst are similar but type can be found by biopsy. They vary in size according to the place from 0.4 inch to 2 inches. They have tenderness on the skin surrounding them there may be swelling and redness as well. However the complete grown cyst is sometimes partially yellow in color.

Sebaceous Cyst Prognosis

The sebaceous cyst when touched or pressed they move to other place. They can be seen anywhere in the body including the genital areas. If the body has problem of producing excessive testosterones the cysts also generated due to that.

Sebaceous Cyst Prevention

The treatment recommended by the doctors is the surgery of that place. it is not certain that after surgery the sebaceous Cyst On Kidney will never appear again because during the surgery the skin is ruptured and the cyst is taken out. Rupture to the organ or the skin is a cause of sebaceous cyst production. It can occur again in the same part even after the successful surgery.

Sebaceous Cyst Treatment

An amazing treatment is the Banical herbal medicine. It is entirely composed of herbal contents that are a result of extensive research from the experts. This medicine is made according to the nature of the body and the response of the skin cells to the sebaceous Cyst On Back. It strengthens the skin cells and makes them strong enough to dissolve the cyst itself. The medicine can be bought from Herbal Care Products. The ingredients of this medicine are Shilajit, Liliaceae, Spiny bamboo, Cloves, Centaurea behen, Cassia, Jadwar, Nutmeg, Early-purple orchid, Himalayan Peony, Red-veined salvia, Ginger, Nux vomica and Grey Amber. It is very effective even in the case of severe issue of cysts. However it is recommended that patient should start using this medicine as the first cyst starts appearing. It also helps in revitalizing the skin when the sebaceous cyst gets ruptured or the mass start coming out of it. This medicine is highly admired by the users and medical specialists.

By : Herbal Care Products

Alopecia – Youthful hair loss problem

What is Alopecia?

What Is Alopecia is the issue of frequent hair loss. It usually occurs due to heredity. It mostly appears because of the ancestors or relatives having the issues. If more than four people in a family have this problem then definitely one person among the generation get affected. Auto immune disorder is an issue of the immune system during which the body works against its own follicles.

Alopecia Causes

The alopecia causes happens when the immune system goes against the hair follicles. The roots of hair get disturbed and weak due to that no further hair growth happens and the already present hair also falls out. Among the infants, causes for alopecia can be diagnosed however certainly it is a bit difficult to treat because their immune system is not properly developed causes of alopecia.

Alopecia symptoms

There are three types of Alopecia Symptoms. The alopecia areata monolocularis , alopecia areata multicularis and ophiasis. During the first one the hair falls in the form of a spot. It happens anywhere on the scalp and results in the complete hair fall on that area. The second one refers to the hair falling from multiple areas that may result in maximum baldness. In the third type of alopecia the hair starts falling in the form of waves and can start from any direction of the head. There are various other sub symptoms of alopecia among them Alopecia areata barbae is the one that results in the hair loss from the beard. However symptoms for alopecia total is the complete hair of scalp falls out and alopecia universalis refers to the hair fall from the whole body including the pubic hair.

Alopecia

Alopecia Diagnosis

Trichoscopy is a method used for the diagnosis of the type of Androgenetic Alopecia areata. Most of the physicians follow various methods for the medication. In very rare cases biopsy is used for the diagnosis of the problem because it does not involve such type of vessels and cysts.

Alopecia Prognosis

The patches of Androgenic Alopecia can easily be recognized when they start whereas the diagnosis methods identify the type when the spots are similar to others. Initially severe itching and inflammation occurs on the areas from where hair fall starts. Usually hair falls from small areas in the start as the time passes these patches start expanding and extreme baldness happens.

Alopecia Prevention

There are various companies who offer oils and other products where as they have different side effects on skin.

Alopecia Treatment

The most reliable medicine for the treatment of alopecia is Apolical. It is highly recommended by the skin and hair specialists for alopecia treatment. It is composed of herbal constituents that include rhubarb, Indian long pepper, fevernut, Calcium sulphate and Gulancha. These are totally natural herbs that prevent the hair fall and increase re-growth. This treatment for alopecia amazingly reduces hair fall and cover the bald patches with new hair. People facing problem of extreme dandruff and excessive hair fall should start using it without wasting time. They can get it from Herbal Care Products. There are amazing results of apolical on the patients no matter from which type of alopecia they are suffering from.

This is the way to treat hair loss. Treatment by alopecia natural remedies we prefer the Herbal Care Products company for patients. alopecia herbal remedies is the apolical. The alopecia natural treatment help to improve hair lossalopecia herbal treatment have no side effects.

By : Herbal Care Products

Abdominal Adhesions – Adhesions related disorder (ARD)

What are Abdominal Adhesions?

Different kinds of fibrous tissues get formulated in result of various factors which tends the internal organs to stick together. They create severe problem inside the body when the intestines fail to move and stretch in case of food digestion. The internal tissues of the body are slippery and help the internal organs to stay separate without sticking together. The Abdominal Adhesions cavity includes the organs from the food pipe that starts after mouth and brings food to the stomach. It also includes different types of intestines and bowel.

Abdominal Adhesions Causes

This issue mostly occurs due to the abdominal surgery. With the passage of time the problems increases and the Abdominal Adhesions Causes gets tighter and increases in number so they also show their harmful effects after years.

Abdominal Adhesions

Abdominal Adhesions Symptoms

There are no specific signs through which they can be diagnosed. The only symptom is the severe Abdominal Adhesions Symptoms pain and cramps in the stomach but they also occurs in many cases. Through other diagnosis methods they are usually located by the physicians. The abdominal X-rays are collected by passing small amount of radiation or the CT scan can be captured. The reports are studied by a radiologist who carefully gives the written diagnosis.

Abdominal Adhesions Diagnosis

During the surgery sometimes other organs get small cuts due to the surgical instruments, they can become one of the reasons for the abdominal Intestinal Adhesions. In some cases the internal organs become dry either due to the drapes used during the surgery or when the surgery goes for longer time. When the internal organs lose their natural moisture and smoothness of their surface they start getting obstructions. They also appear when the blood clots are not removed properly after the surgery. Hence in most of the cases of surgery the abdominal adhesions definitely occur. Although there are various other issues as well due to which they are diagnosed, these are other than the surgical issues. They may include appendix rupture, frequent radiation, gynecological or abdominal infections.

Abdominal Adhesions Prognosis

There is no natural diet which increases or decreases the problem however the doctors advise to take the soft and easily digestible foods. They break easily into smaller particles, due to that they do not get stuck due to the fibers formed in the abdominal cavity.

Abdominal Adhesions Prevention

The cure for this issue is the repeated surgery where as there is no guarantee of preventing them. When the patient undergoes surgery in the abdominal cavity the surgeon arranges some continuous checkups in which they observe the internal condition to prevent the abdominal Uterine Adhesions. They create severe issues if they occur and increase in the body. They can cause infertility among females or due to them the eggs do not locate accurately in the ovary. They can become the reason of miscarriage if the woman has already conceived.

Abdominal Adhesions Treatment

Adhenical is a successful cure for this. This medicine is made from herbal constituents those are highly effective for curing the problem. This abdominal adhesions herbal treatment has no side effects; hence it is highly recommended and should be started as the Abdominal Adhesions Treatment get diagnosed. It is available at Herbal Care Products.

Achalasia – Esophageal Disease

What is Achalasia?

What Is Achalasia. The problem is a very severe health issue that intends the patient to have extreme weight loss due to the problem in swallowing the food and liquids. It sometimes create the infection and swelling in the food pipe that brings food from mouth to stomach called esophagus. When the issues increase and reach an extreme stage it can be converted into chronic diseases.

Achalasia Causes

The food starts getting stuck in the chest and it sticks on several places. Later on it results in creating heart burn and pain. The chronic thing is the excessive weight loss and the pain during the problem. It never lets the patient gain weight and maintains a good health. Ultimately the problem creates many other problems.

Achalasia Symptoms

The problem of Achalasia Symptoms is usually a rare disease that is chronic but is not very common however if one or two symptoms occur in a patient, immediately a physician should be consulted and the patient should start medicine. When the esophagus starts expanding and stretching due to the swelling and issue other problems also start appearing. It should b treated very seriously and carefully because it can cause very harmful effects on the body.

Achalasia

Achalasia Diagnosis

There are several methods through which the diagnosis can be done. The endoscopy of the food pipe is done or the patient goes through the Esophageal Achalasia. The condition is captured through these procedures.

Achalasia Prognosis

It is better that the patient avoid taking the high caffeine products such as coffee ,tea , chips , steaks and other fried items. Patient should have an apple or two in the morning during the problem so that a little energy can be absorbed by the body.

Achalasia Prevention

The patients of Achalasia Cardia are advised to avoid over eating and have reasonable amount of food with no spice and oil. There are many people who use fresh pieces or juices of oranges and grapefruits in breakfast. Physicians and doctors advise such patients to avoid the intake of oranges and other citrus fruits instead of breakfast. The people having this problem should avoid exercise and taking warm milk and eggs before breakfast.

Achalasia Treatment

Achnical is considered as the best Achalasia Treatment. This medicine is composed after the extraordinary research and is made by the medical experts. It has no side effects and helps the maintenance of health without creating any further issues. There are several precautions written on the jar of the medicine. It is extremely focused that the patient facing these issues never stay with empty stomach and the medicine should be taken right after the breakfast. The timing should be strictly followed that is written on the packing or as prescribed by the physician. The achalasia natural treatment helps in balancing the system along with regulating it properly. It is available at the Herbal Care Products. It also reduces the chances of its reappearing and due to no side effects it is highly effective. It only works on the problem so patient does not need any kind of surgery for the remedy.

By : Herbal Care Products

Here’s an interesting article on Delayed Ejaculation!

Natural Herbal Remedies for Delayed Ejaculation

Presenting to you all an article written by a sex therapist on delayed ejaculation as he calls it retarded ejaculation! The writer of this article is Michael A. Perelman, Ph.D from N. Y. Weill Cornell Medical Center, New York, N.Y. USA

Pasting the article here as it is posted on http://www.issm.info/

Retarded ejaculation (RE) is probably the least common, and least understood, of all the male sexual dysfunctions. RE is one of the diminished ejaculatory disorders (DED), which is a subset of male orgasmic disorders (MOD). DED is a collective term for an alteration of ejaculation and /or orgasm that ranges from varying delays in ejaculatory latency to a complete inability to ejaculate, anejaculation, and retrograde ejaculation, as well as reductions in volume, force, and the sensation of ejaculation. The American Psychiatric Association’s Diagnostic and Statistical Manual of Mental Disorders (DSM-IV-TR) defines RE as the persistent or recurrent delay in, or absence of, orgasm after a normal sexual excitement phase during sexual activity that the clinician, taking into account the person’s age, judges to be adequate in focus, intensity, and duration. The disturbance causes marked distress or interpersonal difficulty. Similar to the term “premature ejaculation,” the most commonly used term—“retarded ejaculation”—is sometimes avoided because of its pejorative associations.

In general, RE is reported at low rates in the literature, rarely exceeding 3%. However, based on clinical experiences, some urologists and sex therapists are reporting an increasing incidence of RE. The prevalence of RE appears to be moderately and positively related to age, which is not surprising in view of the fact that ejaculatory function as a whole tends to diminish as men age.

Failure of ejaculation can be a lifelong (primary) or an acquired (secondary) problem. Many men with secondary RE can masturbate to orgasm, whereas others, for multiple reasons, will or cannot. A distinguishing characteristic of men with RE—and one that has implications for treatment—is that they usually have little or no difficulty attaining or keeping their erections—in fact they are often able to maintain erections for prolonged periods of time. Yet, despite their good erections, they report low levels of subjective sexual arousal, at least compared with sexually functional men.
Delayed Ejaculation

Etiology: Biogenic

In some instances, a somatic condition may account for RE, and indeed, any procedure or disease that disrupts sympathetic or somatic innervation to the genital region has the potential to affect ejaculatory function and orgasm. Thus, spinal cord injury, multiple sclerosis, pelvic-region surgery, severe diabetes, and medications that inhibit a-adrenergic innervation of the ejaculatory system have been associated with RE. delayed ejaculation causes, radical prostatectomies (RP) can have a significant negative impact on the sexual function of the patient’s (and partner’s) quality of life. An adequate discussion of the impact of RP on orgasmic capacity is beyond the scope and focus of this article, but suffice it to say a major part of sexual experience and satisfaction has been missing from discussions of sexual rehabilitation following RP. Regrettably, to date, minimal data are available on the post prostatectomy orgasmic experience. While there are anecdotal reports of diminished sensation and pain, Barnas et al. described the only study providing statistics on the prevalence and nature of orgasmic dysfunction after RP. While admirable, this not-yet-replicated study’s sample size (N = 239) was exceedingly modest, given the enormous number of RP procedures performed annually worldwide. Nevertheless, sizable portions of men with RE exhibit no clear somatic factors that account for the disorder.

Pathophysiological causes of RE are far more readily identifiable; they generally surface during a medical history and examination, and they typically stem from predictable sources: anomalous anatomic, neuropathic, endocrine, and medication (iatrogenic). All types of RE show age-related increases in prevalence, independent of increased severity with lower urinary tract delayed ejaculation symptoms. Commonly used medications, particularly antidepressants, may delay ejaculation as well.

Etiology: Psychogenic

Multiple psychosocial explanations have been offered for RE, with unconscious aggression, unexpressed anger, and malingering recurring as themes. In addition, pregnancy fears have been emphasized, as professional referral has often been tied to the female partner’s wish to conceive. Masters and Johnson were the first to suggest an association between RE and religious orthodoxy, positing that certain beliefs limit sexual knowledge and familiarity, causing individuals to “not learn” to ejaculate, or experience an inhibition of normal function.

Recent clinical samples of RE men have noted a disproportionately large number of religiously orthodox/fundamentalist men. Some of these men had very limited sexual knowledge and
masturbated minimally, or not at all. Others, similar to their more secular counterparts,
masturbated for years, but with guilt and anxiety about “spilling seed” subsequently
resulting in RE. The role that religious orthodoxy/fundamentalism plays in the etiology of RE for some menvaries considerably around the world as a function of cultural differences.

Alternatively, men with RE sometimes indicate greater arousal and enjoyment from masturbation than from intercourse. Such an “autosexual” orientation may involve an idiosyncratic and vigorous masturbation style—carried out with high frequency. An “idiosyncratic” masturbation style is one that is not easily duplicated by their partner’s hand, mouth, or vagina. Specifically, many men with RE engage in self-stimulation that is striking in the speed, pressure, duration, and intensity necessary to produce an orgasm, and dissimilar to what they experience with a partner. Disparity between the reality of sex with the partner and the sexual fantasy (whether or not unconventional) used during masturbation is another potential cause of RE.

In short, high-frequency idiosyncratic masturbation, combined with fantasy/partner disparity, may well predispose men to experiencing problems with arousal and ejaculation. This pattern suggests that RE men may lack sufficient levels of physical and/or psychosexual arousal during coitus to achieve orgasm. Inadequate arousal may be responsible for increased anecdotal clinical reports of RE for men using oral medications for the treatment for ED. While most men using phosphodiesterase inhibitors type 5 (PDE-5s) experienced restored erections and coitus with ejaculation, others experienced erection without adequate psycho-emotional arousal. That is, they did not experience sufficient erotic stimulation before and during coitus to reach orgasm, confusing their erect state as an indication of sexual arousal when it primarily indicated vasocongestive success.

Finally, the evaluative/performance aspect of sex with a partner often creates “sexual performance anxiety” for the man, a factor that may contribute to RE. Such anxiety typically stems from the man’s lack of confidence to perform adequately, to appear and feel attractive (body image), to satisfy his partner sexually, to experience an overall sense of self-efficacy, and to measure up against the competition. Anxiety surrounding the inability to ejaculate may draw the man’s attention away from erotic cues that normally serve to enhance arousal. This “ejaculatory performance” anxiety interferes with the erotic sensations of genital stimulation, resulting in levels of sexual excitement and arousal that are insufficient for climax (although more than adequate to maintain an erection).

Evaluation

Retarded ejaculation, then, is best understood as an endpoint or response that represents the interaction of biological, psychological, relationship and cultural factors.

A genitourinary examination and medical history may identify physical anomalies associated with ejaculatory dysfunction as well as contributory neurologic, endocrinologic, or erectile factors. Attention should be given to identifying reversible determinants including: urethral, prostatic, epididymal, testicular infections, and especially insuring the presence of adequate androgen levels. Particularly with secondary RE, adverse pharmaceutical side effects—most commonly from serotonin-based prescriptions—should be ruled out.
A focused psychosexual evaluation is critical and typically begins by differentiating this sexual dysfunction from other sexual problems and reviewing the conditions under which the man is able to ejaculate. The developmental course of the problem—including predisposing issues of religiosity—and variables that improve or worsen performance, particularly those related to psychosexual arousal, should be noted. Perceived partner attractiveness, the use of fantasy during sex, anxiety surrounding performance, and coital and masturbatory patterns all require exploration.

Natural Herbal Remedies of Delayed Ejaculation Treatment

Current sex herbal remedies for delayed ejaculation approaches to RE continue to emphasize the importance of masturbation in the delayed ejaculation treatment of RE. Men who are unaware or unable to become aroused are taught through masturbation and/or partner stimulation to experience increasing levels of arousal through a combination of improved/increased “friction and fantasy.” For others, the focus is on masturbatory retraining, integrated into sex herbal remedies of delayed ejaculation. Masturbation can serve as a type of “dress rehearsal” for sex with a partner. By informing the patient that his difficulty is merely a reflection of “not rehearsing the part he intended to play,” the stigma associated with this problem can be minimized and cooperation of both the patient and partner can be evoked. A man may be encouraged to alter the style of masturbation (“switch hands”) and to approximate (in terms of speed, pressure, and technique) the stimulation likely to be experienced through manual, oral, or vaginal stimulation by his partner.

Of course, masturbation retraining is typically merely a means to an end, and the goal of most current therapeutic techniques for RE (either primary or secondary) is not merely to provide more intense stimulation, but rather to stimulate higher levels of psychosexual arousal and, eventually, orgasm within the framework of a satisfying experience.

Natural remedies of delayed ejaculation for secondary RE follows a strategy similar to that of primary anorgasmia. Men are counseled to use fantasy and bodily movements during coitus, which help approximate the thoughts and sensations perhaps previously experienced only in masturbation. For those individuals the clinician or urologist will often need to counsel these patients to temporarily suspend masturbatory activity and limit orgasmic release to only the desired activity, which is typically coitus.

Partner Issues

To increase satisfactory outcomes from treatment, the partner needs to cooperate with the therapeutic process, finding ways to pleasure the man that enhance arousal and that can be incorporated into the couple’s lovemaking. Sexual fantasies may have to be realigned so that thoughts experienced during masturbation better match those occurring during coitus. Efforts to increase the attractiveness and seductive/arousing capacity of the partner and to reduce the disparity between the man’s fantasy and the actuality of sex with his partner may be useful.

While a number of other partner-related issues may affect a male’s ejaculatory interest and capacity, two require special attention: fertility/conception and anger/resentment.

Regarding conception, the pressure of the woman’s “biological clock” is often the initial treatment driver. The woman—and often the man as well—usually meet any potential intrusion on their plan to conceive with strong resistance. If the urologist or other HCP suspects the patient’s RE is related to fear of conception, he may inquire about the patient’s ability to experience a coital ejaculation with contraception (including condoms) but not during “unprotected” sex. Such a “test” can serve as a powerful diagnostic indicator: if the RE occurs with high probability only during unprotected sex, the HCP can assume that conception is a primary factor causing/maintaining RE.

Whether related to fertility or not, the man’s anger (expressed/unexpressed) toward his partner may be an important intermediate causational factor and must be ameliorated through individual and/or conjoint consultation. Anger is typically a powerful anti-aphrodisiac, and while some men avoid sexual contact entirely when angry at a partner, others attempt to perform, only to find themselves only modestly aroused and unable to maintain an erection/and or reach orgasm. While the man’s assertiveness should be encouraged, the HCP should also remain sensitive and responsive to the impact of this change on the partner—the object of the newly expressed anger—and the resulting alteration in the couple’s equilibrium.

Alternative Treatment Approaches

After natural remedies for delayed ejaculation. While anecdotally viewed by urologists as a difficult-to-treat sexual dysfunction, some sex therapists have reported good success rates, in the neighborhood of 70-80%. Furthermore, although this review has concentrated on the use of counseling methods, there are some medical treatments available.

A number of pharmacological agents have been used off-label to facilitate orgasm in patients taking SSRI antidepressants and other drugs known to delay or inhibit ejaculatory response. Although not approved by regulatory agencies for the treatment of RE, the anti-serotonergic agent cyproheptadine and the dopamine agonist amantadine have been used with moderate success in this population of patients. However, the lack of large, controlled studies with these and other ejaculatory-facilitating agents suggests a high ratio of adverse effects to potential efficacy. Appropriate assessment and consideration of androgen levels is a critical component of treatment. Penile Vibratory Stimulation (PVS) is a potential inexpensive and convenient first-line urological treatment to increase sexual friction and provide the stimulation necessary to restore orgasmic capacity for some who suffered from either a primary or a secondary RE. PVS has been used adjunctively, with discretion, by sex therapists for over thirty years to treat delays/inhibitions of both male and female orgasmic disorders. However, the urological evidence for using PVS, is almost exclusively fertility related; typically for spinal-cord injured men suffering primary RE.

Future Directions: Combination Treatments

Any technique that improves orgasmic response can be usefully incorporated into a combination treatment. Once proof of orgasmic capacity has been reestablished (PVS, for instance), follow-up sessions provide opportunity to increase the probability that orgasm may be evoked by a variety of stimulation techniques, depending on patient and partner preference. As research continues to uncover greater understanding of the ejaculatory process, the likelihood of finding pro-ejaculatory agents increases. As with PE and ED, should safe and effective pharmacological options become available for RE, treatment for this dysfunction will undergo a major paradigm shift, with combination drug and sex therapy protocols likely producing the greatest efficacy and best outcomes in terms of patient satisfaction.

References:

Althof SE, Leiblum SR, Chevert-Measson M, Hartmann U, Levine SB, McCabe M, Plaut M, Rodrigues O, Wylie K. Psychological and interpersonal dimensions of sexual function and dysfunction. In: Lue TF, Basson R, Rosen R, Giuliano F, Khoury S, Montorsi F, eds. Sexual Medicine: Sexual dysfunctions in men and women, pp. 73-115. Paris: Health Publications; 2004.
Association AP. Diagnostic and Statistical Manual of Mental Disorders. Washington, DC: American Psychiatric Association; 2000.
Apfelbaum B. Retarded ejaculation: a much-misunderstood syndrome. In: Leiblum SR, Rosen RC, eds. Principles and Practice of Sex Therapy, pp. 205-241. Guilford Press; 2000.
Blanker MH, Bosch JL, Groeneveld FP, Bohnen AM, Prins A, Thomas S, Hop WC. Erectile and ejaculatory dysfunction in a community-based sample of men 50 to 78 years old: prevalence, concern, and relation to sexual activity. Urology 2001;57:763-768.
Gaulin SJC, McBurney DH. Evolutionary psychology. Upper Saddle River, NJ: Prentice Hall; 2004.
Kaplan HS. The evaluation of sexual disorders: psychologic and medical aspects. New York: Brunner/Mazel; 1995.
Laumann EO, Paik A, Rosen RC. Sexual dysfunction in the United States: prevalence and predictors. JAMA 1999;281:537-544.
Master VA, Turek PJ. Ejaculatory physiology and dysfunction. Urol Clin North Am 2001;28:363-75, x.
Masters WH, Johnson VE. Human Sexual Inadequacy. Boston: Little, Brown & Co.; 1970.
McMahon CG, Abdo C, Incrocci L, Perelman M, Rowland D, Waldinger M, Xin ZC. Disorders of orgasm and ejaculation in men. J Sex Med 2004;1:58-65.
Motofei IG, Rowland DL. Neurophysiology of the ejaculatory process: developing perspectives. BJU Int 2005;96:1333-1338.
Nelson CJ, Ahmed A, Valenzuela R, Parker M, Mulhall JP. Assessment of penile vibratory stimulation as a management strategy in men with secondary retarded orgasm. Urology 2007;69:552-5; discussion 555-6.
Paick JS, Jeong H, Park MS. Penile sensitivity in men with premature ejaculation. Int J Impot Res 1998;10:247-250.
Perelman MA. Masturbation revisited. Contemp Urol 1994;6:68-70.
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Perelman MA. Sex coaching for physicians: combination treatment for patient and partner. Int J Impot Res 2003;15 Suppl 5:S67-74.
Perelman MA. Retarded ejaculation. Current Sexual Health Reports 2004;1:95-101.
Perelman MA. Combination therapy for sexual dysfunction: integrating sex therapy and pharmacotherapy. In: Balon R, Segraves RT, eds. Handbook of Sexual Dysfunction, pp. 13-41. Boca Raton: Taylor & Francis; 2005.
Perelman MA. A new combination treatment for premature ejaculation: a sex therapist’s perspective. J Sex Med 2006;3:1004-1012.
Perelman MA. Abstract #121: The sexual tipping point: a model to conceptualize etiology & combination treatment of female & male sexual dysfunction. J Sex Med 2006;3:52.
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Perelman MA. Idiosyncratic masturbation patterns: a key unexplored variable in the treatment of retarded ejaculation by the practicing urologist. J Urol 2005;173:340, Abstract 1254.
Perelman MA. Unveiling retarded ejaculation. J Urol 2006;175:430, Abstract 1337.
Perelman MA. Integrating sildenafil and sex therapy: unconsummated marriage secondary to ED and RE. J Sex Educ Ther 2001;26:13-21.
Perelman MA, McMahon C, Barada J. Evaluation and treatment of the ejaculatory disorders. In: Lue T, ed. Atlas of Male Sexual Dysfunction, pp. 127-157. Philadelphia: Current Medicine, Inc.; 2004.
Perelman MA, Rowland DL. Retarded ejaculation. World J Urol 2006;24:645-652.
Rosen R, Altwein J, Boyle P, Kirby RS, Lukacs B, Meuleman E, O’Leary MP, Puppo P, Robertson C, Giuliano F. Lower urinary tract symptoms and male sexual dysfunction: the multinational survey of the aging male (MSAM-7). Eur Urol 2003;44:637-649.
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Acknowlegments:
I gratefully thank Dr David L. Rowland who was my co-author on an earlier article upon which this current manuscript was adapted and updated. The original version of this article was published in World Journal of Urology 2006; 24(6): 645-652, Perelman MA, Rowland DL: Retarded Ejaculation. Copyright was kindly granted by Springer Science and Business Media.’ In addition, I also thank Dr Ian Kerner, who graciously provided both editorial guidance and writing assistance for this current version.

Is Atrial Fibrillation going to get worse?

As we’ve already posted many research articles on various disorders this time we found one for Atrial Fibrillation. The question that attracted our attention was “Will Atrial Fibrillation Get Worse?”

We thought of sharing this with those who are reading this right now because you, yes, we knew you are going to find it very helpful. So, here we are posting the article that we found along with the source so that you can read further by going on to the link personally.

The question that was asked was Will Atrial Fibrillation Get Worse?”

They stated that “Early in the disease process, the abnormal heart rhythm often starts and stops in very brief episodes. In people who have pacemakers that record all heartbeats, short runs of atrial fibrillation lasting a few seconds to minutes are often recorded well before symptoms develop. Over time, the episodes may last longer or occur more frequently. Often in this stage of the disease people become aware of the abnormal rhythm. We call this stage of the disease paroxysmal atrial fibrillation.

Studies show that over time, approximately 25-40 percent of people will go on to develop atrial fibrillation that is continuous, called “persistent”. In some of these cases, the heart can often be reset to a normal rhythm with an electrical shock called a cardioversion.

atrial fibrillation

Unfortunately, recurrences of atrial fibrillation after a cardioversion are high. Most people require additional therapies. These may include medicines that control the heart rhythm or ablation procedures to keep the heart in a normal rhythm.

Response to treatment is reduced when a person has persistent atrial fibrillation. In people that remain in paroxysmal atrial fibrillation, Atrial fibrillation natural remedies work better compared to those in persistent atrial fibrillation. For example, a worldwide study of catheter ablation success rates found that 75-83 percent of patients with paroxysmal atrial fibrillation compared to only 63-72 percent in patients with persistent atrial fibrillation remained in a normal rhythm after the procedure – without the need for additional medications. In patients with persistent atrial fibrillation herbal remedies more than one procedure is often required.

The success rates of using medications alone to keep the heart in a normal rhythm are often only 10-40 percent in patients with persistent atrial fibrillation.

Hope you find this read helpful and we will keep coming up with more and more research articles all in one place for you convenience.
***This research article is not by any means our personal asset and we have no right to edit or change it so, whatever we are posting here in the research blog is for information sharing purpose only. ***

 

Source: Everydayhealth

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Breakthroughs in folliculitis!

There are some very informative and appealing articles full of interesting information on folliculitis but we are always on the look for some latest research. We came across this informative excerpt on newsmax.com where there is detailed information on the disease along with some medical breakthroughs explained.

The article is all rights reserved therefore we are posting it here with reference link to the website. We always post articles from various websites to keep out blog updated and make it easy for visitors to find information on one portal but the full article can only be accessed through the original website.

Reference: http://www.newsmax.com/FastFeatures/Folliculitis-treatment-folliculitis-infection/2011/02/09/id/371047/

Here we are just posting the medical breakthroughs mentioned on the website. For any further information you can directly visit the website.

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The website states that “there have been medical breakthroughs in the treatment of folliculitis. Scalp folliculitis infection can be treated by using a natural treatment of folliculitis. Other discoveries include salicylic acid and cade oil scalp cream.

A lotion with aloe vera can be used as a treatment for scalp folliculitis. Scalp folliculitis can also be treated with glycerin lotion. Tomato juice helps reduce sebum levels in the scalp.

The latest treatment for folliculitis is Laser light therapy. It is used to destroy hair follicles and reduce scarring.

Hot, moist compresses promote drainage of extensive folliculitis. In severe cases, carbuncle antibiotics are prescribed which prevent the spread of infection.

Herbal treatment of folliculitis like Turmeric, Tea tree oil, and Apple cider vinegar have been beneficial in treating folliculitis.

These are some of the very helpful tips which can make life easier for a lot of people suffering from this disorder.

Happy reading!!!

Some Treatment Options for Scleroderma!

Hello all! Here’s an informative article on Scleroderma which we came across while doing some research on the disorder. We found it informative and thought we should share it with you all. We will be; from time to time keep posting informative articles for your reading.
This article has been extracted from http://www.hopkinsscleroderma.org/ on Scleroderma treatment options. This is a selection from the following source:
Chapter 23 of Systemic Sclerosis,
2nd Edition
written by

Dr. Laura Hummers and Dr. Fred Wigley.

Because no two cases of Scleroderma are alike, identifying your disease subtype, stage, and involved organs is very important in determining the best course of action for treatment. Current therapies use medications that focus on the four main features of the disease: inflammation, autoimmunity, vascular disease, and tissue fibrosis. Your physician will work with you to identify the treatments that are best for you, but here are some common treatment options:

ANTI-INFLAMMATORY MEDICATIONS

Many medications are thought to directly or indirectly affect inflammation. In scleroderma, there are two major types of inflammation that are related to the disease process. The first is a more conventional type that can cause arthritis (inflammation in the joints), myositis (inflammation in the muscles), or serositis [inflammation in the lining of the heart (pericarditis) or lining of the lung (pleuritis)]. This type of inflammation responds to traditional antiinflammatory drugs: NSAIDs (e.g. ibuprofen) or corticosteroids (e.g. prednisone). The duration of therapy and the dose of medication are dictated by the specific situation. Some patients will need chronic administration and others will recover after a limited course of therapy.
The other type of inflammation relates to the skin and other tissue injury caused by the scleroderma process. This phase of the disease does not appear to respond to NSAIDs or corticosteroids, although the exact role of corticosteroids is not fully studied. There are risks associated with the use of these agents, including gastrointestinal disease, fluid retention, and renal toxicity. Corticosteroid use is also associated with an increased risk of scleroderma symptoms renal crisis. Therefore, it is recommended that the use of NSAIDs and corticosteroids be limited to inflammatory states that demonstrate responsiveness.

IMMUNOSUPPRESSIVE THERAPY

The most popular approach to controlling the inflammatory phase of scleroderma is the use of immunosuppressive therapy. The rationale is that an autoimmune process is causing the inflammation and the downstream result is tissue damage and fibrosis. In this model, the fibrosis is an “innocent bystander” that is driven by the cytokines (chemical messengers) produced by the immune system. There are several drugs that are being used, but only a few well designed studies have been performed. These immunosuppressing drugs include methotrexate, cyclosporine, antithymocyte globulin, mycophenolatemofetil and cyclophosphamide. A recent study suggested that methotrexate did not significantly alter the skin score (a measure of skin thickening) compared with placebo (no treatment). Cyclosporine is not completely studied due to reports of renal toxicity. The most promising drugs are mycophenolatemofetil or cyclophosphamide with or without antithymocyte globulin. Unfortunately, there is no placebo-controlled study (i.e., half the patients get the medication and half get a sugar pill) to define their exact role in treating scleroderma causes, but if used during the active inflammatory phase of the disease, they appear to work.
A major area of current research is the use of aggressive immunosuppressive therapy either with very-high-dose cyclophosphamide or with autologous bone marrow transplantation. Because these aggressive forms of immunosuppressive therapy have potential risks, they should be used in severe cases of scleroderma and administered as part of a research protocol.

Scleroderma

DRUG THERAPY OF VASCULAR DISEASE

The vascular disease in scleroderma is widespread and affects medium and small arteries. It is manifest clinically as Raynaud’s phenomenon in the skin, and there is evidence that repeated episodes of ischemia (low-oxygen state) occur in other tissues. Low blood flow into the skin and tissues is thought not only to damage tissue by the lack of nutrition and oxygen but to activate fibroblasts and promote tissue fibrosis. Therefore, treatment of the vascular disease is now considered crucial to controlling the disease as a whole as well as preventing specific organ damage. There are three major features of the vascular disease that potentially need treatment: vasospasm (spasm of blood vessels), a proliferative vasculopathy (thickening of blood vessels), and thrombosis (blood clots) or structural occlusion of the vessel lumen (blockage of blood vessels).
Vasospasm is best treated with vasodilator therapy (drugs that open blood vessels). The most effective and popular vasodilator therapy continues to be the calcium channel blockers (e.g., nifedipine). Studies demonstrate that the calcium channel blockers can reduce the frequency of Raynaud’s phenomenon attacks and reduce the occurrence of digital ulcers. It is now known that the microcirculation of each organ has a unique mechanism for controlling its own blood supply. The skin blood flow is regulated by the sympathetic nervous system; the kidney blood flow by locally produced hormones such as renin; and the circulation in the lung by endothelin, prostaglandins and nitric oxide. There are very specific agents to counteract the negative influence of the scleroderma vascular disease on each involved organ. For example, the calcium channel blockers are reported to help blood flow to the skin and heart; angiotensin converting enzyme inhibitors (ACE) inhibitors reverse the vasospasm of the scleroderma renal crisis; and bosentan (a new endothelin-1 receptor inhibitor) or epoprostenol (prostacyclin) improve blood flow in the lung.
Although there are several vasoactive drugs on the market that are being used to treat vascular disease, there is no agent that is known to reverse the intimal proliferation (thickening of the inner layer of the blood vessel) that is part of the scleroderma vascular disease. Drugs that reverse vasospasm (calcium channel blockers, bosentan, prostacyclin, or nitric oxide) all have the potential to modify the course of the disease. There is evidence that these vasodilators may also directly affect the tissue fibrosis. For example, bosentan may be of benefit because it inhibits endothelin-1, a molecule produced by blood vessels that can also directly activate tissue fibroblasts to make collagen.
The final outcome of untreated scleroderma vascular disease is occlusion of the vessels by either thrombus formation or advanced fibrosis of the intima. Therefore, anti-platelet therapy in the form of low-dose aspirin is recommended. Good studies to determine if antiplatelet or anticoagulation therapy is helpful do not exist. In an acute digital ischemic crisis (sudden development of threatened loss of a digit), anti-coagulation (use of blood-thinning medications) is often used for a short period.

ANTI-FIBROTIC AGENTS

It has been know for years that, in scleroderma, excess collagen is being produced in the skin and other organs. Several drugs are used that have in vitro (in the tissue culture) ability to reduce collagen production or to destabilize tissue collagen. The older medications in this category include colchicine, para-aminobenzoic acid (PABA), dimethyl sulfoxide, and D-penicillamine. Although there is evidence for and against the use of these agents, most experts are disappointed with them and believe that the benefit either does not exist or the drug is not potent enough to warrant its use. D-penicillamine remains a popular alternative for some experts, despite a controlled trial demonstrating no difference between low and high doses of the drug.
The search for new drugs that alter the fibrotic reaction is probably one of the most active areas of scleroderma research. Strategies include directly suppressing the fibroblast and its ability to make collagen, inhibiting the cytokines that activate the fibroblast, and the use of agents that might break down collagen faster and promote tissue remodeling.
If you want full reading you can go to chapter 23 of Systemic Sclerosis by clicking the following link:
http://www.hopkinsscleroderma.org/downloads/SystemicSclerosis_Chapter23.pdf
Happy reading!

What research is being done on Benign Essential Tremor?

While doing a research on Benign Essential Tremor we came across this article on NINDS web portal. The title ‘What Research is Being Done?’ caught our attention. Here we are pasting the same article or let’s say the para that was of interest to us. There is also define Benign Essential Tremor Symptoms.

What research is being’ done?

The Benign Essential Tremor Causes. The website state that “The National Institute of Neurological Disorders and Stroke, a unit of the National Institutes of Health (NIH) within the U.S. Department of Health and Human Services, is the nation’s leading federal funder of research on disorders of the brain and nervous system. The NINDS sponsors research on tremor both at its facilities at the NIH and through grants to medical centers.

Benign Essential Tremor

Scientists at the NINDS are evaluating the effectiveness of 1-octanol, a substance similar to alcohol but less intoxicating, for treating essential tremor. Results of two previous NIH studies have shown this agent to be promising as a potential new Benign Essential Tremor Treatment.

Scientists are also studying the effectiveness of botulinum toxin as a treatment for a variety of involuntary movement disorders, including essential tremor of the hand.”

That is it for now. We will be back with some new research soon because we love to share what we find.

There are some tips that work for Sebaceous Cyst

From different resources online and offline we came up with some very helpful tips which can help a great deal in dealing with sebaceous cysts but they are not tried and tested personally by any of our member. These have been collected from various resources as mentioned earlier. To find out read on!

  •  Using castor oil reduces the occurrence of sebaceous cyst.
  • Tea tree oil is also very beneficial in taking care of sebaceous cysts.
  •  You can make a bandaid by mixing castor oil and tea tree oil and apply on the area covered by cysts.
  • White vinegar injection has also been very successful in getting rid of the sebaceous cysts.
  •  Make a paste of turmeric and apply on the affected areas you sure are going to see amazing results that’s what a lot of  people claim.
  • Heat has also been very helpful in reducing the size of these cysts to completely erasing them.
  •  Applying Ichthammol ointment on the cysts at night before sleeping has also shown great results. Buy it from any pharmacy around you.
  • Changing dietary routine can be of immense importance and can play a key role in helping you get rid of these cysts.
  • Increase the intake of iodine which a lot of people have reported has worked for them.
  •  Reduce gluten in your diet and you can prevent more cysts from forming.
  •  Reduce the intake of refined sugar which has been identified as the main culprit in triggering the onset of sebaceous cysts.

Sebaceous Cyst

These are all some of the useful tips which can be of great help and can save you from going through a surgical procedure. Try these techniques at your own risk. Herbal Care Products is not responsible for any damage caused due to these tips. These are for information purpose only.